Individualizing therapy for pancreatic cancer

Emily K. Colvin; David K. Chang; Neil D. Merrett; James G. Kench; Andrew V. Biankin

doi:10.1111/j.1440-1746.2008.05683.x

Individualizing therapy for pancreatic cancer

Emily K. Colvin, David K. Chang, Neil D. Merrett, James G. Kench, Andrew V. Biankin

Research output: Contribution to journal › Article › peer-review

6 Citations (Scopus)

Abstract

Editorial. See article in J. Gastroenterol. Hepatol. 2008; 23: 1852–1859 The study by Jiang and colleagues in this issue of the Journal of Gastroenterology and Hepatology reports the relationship of neural growth factor (NGF), tyrosine kinase receptor A (TrkA), and perineural invasion in pancreatic cancer (PC). They hypothesize that these molecules may play a key role in perineural invasion, and hence, tumor progression and metastases. As a consequence, they may be clinically useful markers (biomarkers) of poor prognosis and potential targets for the development of novel therapeutic strategies.

Original language	English
Pages (from-to)	1779-1782
Number of pages	4
Journal	Journal of Gastroenterology and Hepatology
Volume	23
Issue number	12
DOIs	https://doi.org/10.1111/j.1440-1746.2008.05683.x
Publication status	Published - 2008

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10.1111/j.1440-1746.2008.05683.x

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title = "Individualizing therapy for pancreatic cancer",

abstract = "Editorial. See article in J. Gastroenterol. Hepatol. 2008; 23: 1852–1859 The study by Jiang and colleagues in this issue of the Journal of Gastroenterology and Hepatology reports the relationship of neural growth factor (NGF), tyrosine kinase receptor A (TrkA), and perineural invasion in pancreatic cancer (PC). They hypothesize that these molecules may play a key role in perineural invasion, and hence, tumor progression and metastases. As a consequence, they may be clinically useful markers (biomarkers) of poor prognosis and potential targets for the development of novel therapeutic strategies.",

author = "Colvin, {Emily K.} and Chang, {David K.} and Merrett, {Neil D.} and Kench, {James G.} and Biankin, {Andrew V.}",

year = "2008",

doi = "10.1111/j.1440-1746.2008.05683.x",

language = "English",

volume = "23",

pages = "1779--1782",

journal = "Journal of Gastroenterology and Hepatology",

issn = "0815-9319",

publisher = "John Wiley & Sons",

number = "12",

}

TY - JOUR

T1 - Individualizing therapy for pancreatic cancer

AU - Colvin, Emily K.

AU - Chang, David K.

AU - Merrett, Neil D.

AU - Kench, James G.

AU - Biankin, Andrew V.

PY - 2008

Y1 - 2008

N2 - Editorial. See article in J. Gastroenterol. Hepatol. 2008; 23: 1852–1859 The study by Jiang and colleagues in this issue of the Journal of Gastroenterology and Hepatology reports the relationship of neural growth factor (NGF), tyrosine kinase receptor A (TrkA), and perineural invasion in pancreatic cancer (PC). They hypothesize that these molecules may play a key role in perineural invasion, and hence, tumor progression and metastases. As a consequence, they may be clinically useful markers (biomarkers) of poor prognosis and potential targets for the development of novel therapeutic strategies.

AB - Editorial. See article in J. Gastroenterol. Hepatol. 2008; 23: 1852–1859 The study by Jiang and colleagues in this issue of the Journal of Gastroenterology and Hepatology reports the relationship of neural growth factor (NGF), tyrosine kinase receptor A (TrkA), and perineural invasion in pancreatic cancer (PC). They hypothesize that these molecules may play a key role in perineural invasion, and hence, tumor progression and metastases. As a consequence, they may be clinically useful markers (biomarkers) of poor prognosis and potential targets for the development of novel therapeutic strategies.

UR - http://handle.uws.edu.au:8081/1959.7/535820

U2 - 10.1111/j.1440-1746.2008.05683.x

DO - 10.1111/j.1440-1746.2008.05683.x

M3 - Article

SN - 0815-9319

VL - 23

SP - 1779

EP - 1782

JO - Journal of Gastroenterology and Hepatology

JF - Journal of Gastroenterology and Hepatology

IS - 12

ER -

Individualizing therapy for pancreatic cancer

Abstract

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