Tumor necrosis factor (TNF) inhibitors

Kevin D. Pile; Garry G. Graham; Stephen M. Mahler

doi:10.1007/978-3-7643-8550-7_67

Tumor necrosis factor (TNF) inhibitors

Kevin D. Pile, Garry G. Graham, Stephen M. Mahler

Western Sydney University

Research output: Chapter in Book / Conference Paper › Chapter

Abstract

![CDATA[The tumor necrosis factor (TNF) inhibitors are protein constructs which bind and inactivate TNF. They are members of a group of proteins commonly termed biological response modifiers (BRMs) or biological disease-modifying antirheumatic drugs (bDMARDs). They are not included in the group, slow-acting antirheumatic drug (SAARDs), because they are considered to have more specific actions and because their therapeutic actions are produced rapidly. The TNF inhibitors are active in the treatment of rheumatoid arthritis, while most also improve psoriatic arthritis, ankylosing spondylitis, ulcerative colitis, and Crohn’s disease.]]

Original language	English
Title of host publication	Compendium of Inflammatory Diseases
Editors	Michael J. Parnham
Place of Publication	Switzerland
Publisher	Springer
Pages	1282-1290
Number of pages	9
ISBN (Electronic)	9783764385507
ISBN (Print)	9783764385309
DOIs	https://doi.org/10.1007/978-3-7643-8550-7_67
Publication status	Published - 2016

Keywords

tumor necrosis factor
biological response modifiers
antirheumatic agents

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10.1007/978-3-7643-8550-7_67

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title = "Tumor necrosis factor (TNF) inhibitors",

abstract = "![CDATA[The tumor necrosis factor (TNF) inhibitors are protein constructs which bind and inactivate TNF. They are members of a group of proteins commonly termed biological response modifiers (BRMs) or biological disease-modifying antirheumatic drugs (bDMARDs). They are not included in the group, slow-acting antirheumatic drug (SAARDs), because they are considered to have more specific actions and because their therapeutic actions are produced rapidly. The TNF inhibitors are active in the treatment of rheumatoid arthritis, while most also improve psoriatic arthritis, ankylosing spondylitis, ulcerative colitis, and Crohn{\textquoteright}s disease.]]",

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AU - Pile, Kevin D.

AU - Graham, Garry G.

AU - Mahler, Stephen M.

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AB - ![CDATA[The tumor necrosis factor (TNF) inhibitors are protein constructs which bind and inactivate TNF. They are members of a group of proteins commonly termed biological response modifiers (BRMs) or biological disease-modifying antirheumatic drugs (bDMARDs). They are not included in the group, slow-acting antirheumatic drug (SAARDs), because they are considered to have more specific actions and because their therapeutic actions are produced rapidly. The TNF inhibitors are active in the treatment of rheumatoid arthritis, while most also improve psoriatic arthritis, ankylosing spondylitis, ulcerative colitis, and Crohn’s disease.]]

KW - tumor necrosis factor

KW - biological response modifiers

KW - antirheumatic agents

UR - http://handle.westernsydney.edu.au:8081/1959.7/uws:40023

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DO - 10.1007/978-3-7643-8550-7_67

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SP - 1282

EP - 1290

BT - Compendium of Inflammatory Diseases

A2 - Parnham, Michael J.

PB - Springer

CY - Switzerland

ER -

Tumor necrosis factor (TNF) inhibitors

Abstract

Keywords

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